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Chapter_28:_Gastrointestinal_Hormones
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07/07/2024
Stomach acid secretion is stimulated by {{c1::gastrin}} from G cells of the stomach antrum
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07/07/2024
The inhibitory hormone of the gastrointestinal system is {{c1::somatostatin}} from D cells in the stomach antrum
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The hormone responsible for pancreatic enzyme secretion is {{c1::cholecystokinin}} from I cells in the duodenum
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The hormone responsible for HCO3 release is {{c1::secretin}} from S cells of the duodenum
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High pancreatic duct output results in {{c1::high}} HCO3 output
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Slow pancreatic duct output results in {{c1::low}} HCO3 output
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Vasoactive intestinal peptide (VIP) results in increased intestinal secretions and {{c1::motility}}
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The hormone released in a starvation state is {{c1::glucagon}} from alpha cells in the pancreas
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The hormone released in a fed state is {{c1::insulin}} from beta cells in the pancreas
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Pancreatic polypeptide results in {{c1::decreased}} pancreatic and gallbladder secretions(increased or decreased)
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Motilin is primarily released from the duodenum and acts on the antrum to {{c1::increase}} intestinal motility(increase or decrease)
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Gastrin-releasing peptide (GRP) is also known as {{c2::bombesin}} and results in {{c1::increased}} intestinal motor, enzyme, and acid secretion
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Peptide YY from the terminal ileum causes {{c1::decreased}} acid secretion and stomach contraction(increased or decreased)
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The fastest bowel recovery time is with the {{c1::small bowel}}
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The slowest bowel recovery time is with the {{c1::large bowel}}
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Resting is peristalsis phase {{c1::1}}
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Accelerating is peristalsis phase {{c1::2}}
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Peristalsis is peristalsis phase {{c1::3}}
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07/07/2024
Decelerating is peristalsis phase {{c1::3}}
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