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Chapter_16:_Critical_Care
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Published
07/07/2024
The normal value for cardiac output (CO) is {{c1::4}} to {{c1::8}} L/min
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The normal value for cardiac index (CI) is {{c1::2.5}} to {{c1::4}} L/min
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The normal value for systemic vascular resistance (SVR) is {{c1::800}} to {{c1::1400}} dynes/second/cm-5
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The normal value for pulmonary capillary wedge pressure (PCWP) is {{c1::7}} to {{c1::15}} mmHg
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The normal value for central venous pressure (CVP) is {{c1::5}} to {{c1::9}} mmHg
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The normal value for pulmonary artery pressure (PAP) is {{c1::25}}/{{c1::10}} mmHg
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The normal value for mixed venous oxygen saturation (SvO2) is {{c1::70}} to {{c1::80}}%
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Cardiac index (CI) can be calculated with the equation:{{c1::cardiac output (CO)}} / {{c1::body surface area (BSA)}}
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The kidney receives {{c1::25}}% of the cardiac output
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The brain receives {{c1::15}}% of the cardiac output
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The heart receives {{c1::5}}% of the cardiac output
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Cardiac preload can be measured by finding the {{c1::pulmonary capillary wedge pressure (PCWP)}}
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Cardiac afterload can be measured by finding the {{c1::systemic vascular resistance (SVR)}}
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Cardiac output increases with heart rate, but plateaus due to decreased {{c1::diastolic filling time}}
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The atrial kick accounts for {{c1::20}}% of left ventricular end diastolic volume (LVEDV)
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An automatic increase in contractility secondary to increased afterload is known as the {{c1::Anrep}} effect
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An automatic increase in contractility secondary to increased heart rate is known as the {{c1::Bowditch}} effect
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Arterial O2 content (CaO2) is calculated with the equation:{{c1::Hgb}} x {{c1::1.34}} x {{c1::O2 saturation}} + ({{c2::PO2}} x {{c2::0.003}}…
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O2 delivery is calculated with the equation:{{c1::CO}} x {{c2::CaO2}} x 10
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O2 consumption is calculated with the equation:{{c2::CO}} x ({{c1::CaO2}} - {{c1::CvO2}})
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The normal O2 delivery-to-consumption ratio is {{c1::4}} to {{c1::1}}
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The normal pressure of oxygen where 50% of O2 receptors are saturated (p50) is {{c1::27}} mmHg
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Shunting of blood and decreased O2 extraction cause a(n) {{c1::increase}} in SvO2(increase or decrease)
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Increased O2 extraction and decreased O2 delivery cause a(n) {{c1::decrease}} in SvO2(increase or decrease)
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A Swan-Ganz catheter should be placed in zone {{c1::III}} of the lung to measure PCWP
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Hemoptysis after flushing a Swan-Ganz catheter should be initially managed with {{c1::increasing PEEP}}
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An absolute contraindication to Swan-Ganz catheter use is a(n) {{c1::right-sided mechanical valve}}
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The approximate Swan-Ganz catheter distance to wedge from the right subclavian vein is {{c1::45}} cm
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The approximate Swan-Ganz catheter distance to wedge from the right internal jugular vein is {{c1::50}} cm
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The approximate Swan-Ganz catheter distance to wedge from the left subclavian vein is {{c1::55}} cm
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The approximate Swan-Ganz catheter distance to wedge from the left internal jugular vein is {{c1::60}} cm
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Can echocardiography measure pulmonary vascular resistance (PVR)?{{c1::No}}
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Wedge pressure measurements should be taken at {{c1::end-expiration}}
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The partial pressure of oxygen of blood in the left ventricle is {{c1::lower}} than in the pulmonary capillaries(higher or lower)
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The normal alveolar-arterial gradient in a non-ventilated patient is {{c1::10}}-{{c1::15}} mmHg
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Blood with the lowest venous saturation is blood from the {{c1::coronary sinus (30%)}}
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Blood with the highest venous saturation is blood from the {{c1::renal veins (80%)}}
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Inadequate tissue oxygenation is the most basic definition of {{c1::shock}}
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The most common cause of adrenal insufficiency is {{c1::withdrawal of exogenous steroids}}
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The medical treatment of adrenal insufficiency {{c1::dexamethasone}}
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Cortisone and hydrocortisone have the {{c1::lowest}} relative steroid potency (lowest or highest)
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Dexamethasone has the {{c1::highest}} relative steroid potency (lowest or highest)
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Loss of sympathetic tone is characteristic of {{c1::neurogenic}} shock
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Treatment for neurogenic shock is {{c1::volume resuscitation}} first and then {{c2::phenylephrine}}
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The initial presentation of hemorrhagic shock is increased {{c1::diastolic pressure}}
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Cardiogenic shock is treated with {{c2::dobutamine}} and {{c1::intra-aortic balloon pump (IABP)}}
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Cardiac tamponade causes hypotension due to decreased {{c1::ventricular filling}}
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The early sepsis triad consists of {{c2::hyper}}-ventilation, confusion, and {{c1::hypo}}-tension
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Early gram-negative sepsis has {{c1::decreased}} insulin
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Late gram-negative sepsis has {{c1::increased}} insulin
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Epinephrine and norepinephrine are released in hypovolemia in the {{c1::rapid}} neurohormonal response(rapid or sustained)
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Renin, ADH, and ACTH are released in hypovolemia in the {{c1::sustained}} neurohormonal response(rapid or sustained)
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Petechia, hypoxia, and confusion are characteristic of {{c1::fat}} emboli
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Intubated patients with pulmonary emboli can present with {{c1::hypo}}-tension and {{c2::decreased::increased or decreased}} ETCO2
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The most common EKG finding in pulmonary embolism is {{c1::tachycardia}}
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Most pulmonary embolisms arise from the {{c1::iliofemoral}} region
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Central lines or pulmonary artery catheters can be used to aspirate {{c1::air}} emboli out of the right heart
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Intra-aortic balloon pumps inflate during {{c1::dia}}-stole
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Intra-aortic balloon pumps deflate during {{c1::sys}}-stole
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Intra-aortic balloon pumps inflate on the {{c1::T}} wave
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Intra-aortic balloon pumps deflate on the {{c1::P}} wave
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Intra-aortic balloon pumps can be used for {{c1::cardiogenic}} shock
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Intra-aortic balloon pumps can be used in patients with refractory {{c1::angina}} awaiting revascularization
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Intra-aortic balloon pumps {{c1::decrease}} afterload (increase or decrease)
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Intra-aortic balloon pumps {{c1::increase}} diastolic blood pressure(increase or decrease)
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Receptors free card{{c1:::)}}You should know this already from med school
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Dopamine at 2-5 micrograms/kg/min mainly acts as a(n) {{c1::dopamine}} receptor agonist
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Dopamine at 6-10 micrograms/kg/min mainly acts as a(n) {{c1::beta-adrenergic}} receptor agonist
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Dopamine at >10 micrograms/kg/min mainly acts as a(n) {{c1::alpha-adrenergic}} receptor agonist
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Dobutamine acts as a(n) {{c1::beta-1}} receptor agonist
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Milrinone acts as an inhibitor of {{c1::phosphodiesterase}}
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Phenylephrine acts as a(n) {{c1::alpha-1}} receptor agonist
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Norepinephrine acts as a(n) {{c1::alpha-1, alpha-2, and beta-1}} receptor agonist
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Epinephrine at a low dose acts as a(n) {{c1::beta-1 and beta-2}} receptor agonist
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Epinephrine at a high dose acts as a(n) {{c1::alpha-1 and alpha-2}} receptor agonist
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Isoproterenol acts as a(n) {{c1::beta-1 and beta-2}} receptor agonist
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Vasopressin causes arterial vasoconstriction when it acts on {{c1::V1}} receptors
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Vasopressin causes water resorption at the collecting ducts when it acts on {{c1::intrarenal V2}} receptors
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Vasopressin causes release of factor VIII and von Willebrand factor when it acts on {{c1::extrarenal V2}} receptors
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Cyanide toxicity with sodium nitroprusside use can be monitored by measuring {{c1::thiocyanate}} levels
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Nitroglycerin predominantly works by reducing {{c1::pre}}-load
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Change in lung volume divided by change in lung pressure is the equation for lung {{c1::compliance}}
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FEV1 and vital capacity tend to {{c1::decrease}} with aging(increase or decrease)
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Functional residual capacity (FRC) tends to {{c1::increase}} with aging(increase or decrease)
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The ventilation to perfusion ratio is highest in the {{c1::upper}} lobes of the lung(upper or lower)
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The ventilation to perfusion ratio is lowest in the {{c1::lower}} lobes of the lung(upper or lower)
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Increasing PEEP on the ventilator tends to {{c1::increase}} function residual capacity (FRC)(increase or decrease)
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Increasing rate or tidal volume on the ventilator tends to {{c1::decrease}} CO2(increase or decrease)
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To wean off the ventilator, FiO2 should be < {{c1::40}}%
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To wean off the ventilator, PEEP should be {{c1::5}}
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To wean off the ventilator, respiratory rate should be < {{c1::24}} per min
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To wean off the ventilator, heart rate should be < {{c1::120}} beats per min
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To wean off the ventilator, PO2 should be > {{c1::60}} mmHg
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To wean off the ventilator, PCO2 should be < {{c1::50}} mmHg
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To wean off the ventilator, O2 saturation should be > {{c1::93}}%
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A ventilation setting that decreases the the work of breathing by providing constant inspiratory pressure is {{c1::pressure}} support
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FiO2 should be kept below {{c1::60}}% to prevent O2 radical toxicity to the lungs
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Excessive PEEP can cause decreased cardiac output mainly due to decreased {{c1::right atrial filling}}
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High-frequency ventilation is used more commonly in {{c1::pediatrics}}(pediatrics or adults)
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Free card {{c1:::)}}All of this information should be review. Not gonna make any cards over it, but if you think you need to you can. I will just leav…
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Parts of lung that are ventilated but not perfused are called {{c1::dead space}}
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Parts of lung that are perfused but not ventilated are called {{c1::shunt}}
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The most common cause of increased dead space is {{c1::excessive PEEP}}
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The most common cause of increased shunt is {{c1::atelectasis}}
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Increased dead space tends to lead to {{c1::hypercapnia}}(hypoxia or hypercapnia)
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Increased shunting tends to lead to {{c1::hypoxia}}(hypoxia or hypercapnia)
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The most common cause of ARDS is {{c1::pneumonia}}
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The treatment of ARDS involves decreasing barotrauma by allowing permissive {{c1::hypercapnia}}
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The treatment of ARDS involves using {{c1::lower}} tidal volumes(higher or lower)
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The treatment of ARDS involves {{c1::increasing}} the inspiratory time(increasing or decreasing)
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The diagnostic criteria for Acute Respiratory Distress Syndrome (ARDS) involves {{c1::acute}} onset
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The diagnostic criteria for Acute Respiratory Distress Syndrome (ARDS) involves {{c1::bilateral}} pulmonary in…
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The diagnostic criteria for Acute Respiratory Distress Syndrome (ARDS) involves PaO2 / FiO2 of < …
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The diagnostic criteria for Acute Respiratory Distress Syndrome (ARDS) involves absence of {{c1::heart failure}}
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Chemical pneumonitis from aspiration of gastric secretions is known as {{c1::Mendelson's}} syndrome
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The most frequent site of aspiration pneumonia is the {{c1::superior}} segment of the right {{c2::lower}} lobe
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Post-operatively, atelectasis is usually caused by poor {{c1::inspiration}}
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The most common cause of fever in the first 48 hours after operation is {{c1::atelectasis}}
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The most common cause of pulmonary vasoconstriction is {{c1::hypoxia}}
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Alkalosis causes pulmonary vaso-{{c1::dilation}}
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Acidosis causes pulmonary vaso-{{c1::constriction}}
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The most common cause of poor urine output early post-op is {{c1::hypovolemia}}
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The most common cause of post-operative renal failure is intra-operative {{c1::hypotension}}
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The best test for azotemia is {{c1::fractional excretion of sodium (FENa)}}
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The 1st step in management for oliguria is to make sure the patient is {{c1::volume loaded}}
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The 2nd step in management for oliguria is a trial of {{c1::furosemide}}
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The 3rd step in management for oliguria is {{c1::dialysis}}
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The treatment of pre-renal oliguria is {{c1::fluids}}
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The treatment of intra-renal oliguria is {{c1::diuretic trial}}
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The treatment of post-renal oliguria is {{c1::relieve the obstruction}}
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Hemo-{{c1::dialysis}} tends to be faster and can cause large volume shifts
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Hemo-{{c1::filtration}} tends to be slower and does not cause large volume shifts
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Angiotensin II causes vasoconstriction and acts as a(n) {{c1::inhibitor}} of renin release
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Atrial natriuretic peptide (ANP) acts as a vaso-{{c1::dilator}}
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Antidiuretic hormone (ADH) is released from the {{c1::posterior}} pituitary when osmolality is high
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NSAIDs cause renal damage by {{c1::inhibiting prostaglandin synthesis}}
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Aminoglycosides, myoglobin, and contrast dyes all cause renal damage by {{c1::direct tubular injury}}
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Renal injury from contrast dyes are treated with {{c1::pre-hydration}} before exposure
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The most potent stimulus for SIRS is {{c1::endotoxin}}
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Sepsis is defined as SIRS with a(n) {{c1::infection}}
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Septic shock is defined as sepsis with {{c1::hypotension}}
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Brain death cannot be diagnosed if the patient temperature is below {{c1::32}} degrees Celsius
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Brain death cannot be diagnosed if the patient blood pressure is below {{c1::90}} mmHg
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An EEG in brain death shows {{c1::electrical silence}}
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An MRA in brain death shows {{c1::no blood flow to brain}}
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A positive apnea test will show a CO2 level of greater than {{c1::60}} mmHg or an increase of {{c2::20}} mmHg
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Can patients with brain death still have deep tendon reflexes?{{c1::Yes}}
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Patients with carbon monoxide poisoning can have a(n) {{c1::high}} O2 saturation on standard pulse oximeter
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Carbon monoxide poisoning usually corrects when treated with {{c1::100% oxygen on ventilator}}
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Methemoglobinemia tends to give an O2 saturation value of {{c1::85}}%
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Cyanide toxicity tends to present as a {{c1::left}}-to-{{c1::right}} shunt
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Critical illness polyneuropathy tends to present more as a {{c1::motor}} neuropathy(motor or sensory)
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Xanthine oxidase in endothelial cells can form oxygen radicals, which is involved in {{c1::reperfusion injury}}
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The initial treatment for diabetic ketoacidosis (DKA) is {{c1::normal saline}} and insulin
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Alcohol withdrawal is treated with the vitamins {{c1::thiamine}}, {{c1::folate}}, and {{c1::B12}}
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Alcohol withdrawal is treated with the electrolytes {{c1::magnesium}} and {{c1::potassium}}
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Alcohol withdrawal is treated with PRN {{c1::lorazepam::drug}}
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ICU psychosis general occurs after post operative day {{c1::3}}
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The most common cause of delayed discharge after cardiac surgery is {{c1::atrial fibrillation}}
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Patients with suspected sepsis should recieve fluids at {{c1::30}} mL/kg over the first {{c1::3}} hours
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Hypotensive skin graft patients should get {{c1::vasopressin::pressor}} before {{c1::norepinephrine::pressor}}
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Aggressive fluid resuscitation before a sepsis diagnosis is confirmed is controversial because many patients end up being {{c1::fluid overloaded}} des…
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A positive central line blood culture with a negative peripheral blood culture should be treated {{c1::as a contaminant}}
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In suspected sepsis, early administration of {{c1::antibiotics::fluids or abx}} is {{c2::more::more or less}} important than early administration…
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SIRS criteria - Temperature: {{c1::>100.4 or <96.8}} - Respiratory Rate: {{c2::>20}} - Heart Rate: {{c3::>90}} - WBC: …
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Sepsis diagnosis is {{c2::2 SIRS criteria}} and {{c1::suspected or confirmed source of infection}}
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Severe sepsis is {{c2::sepsis}} and {{c1::hypotension <90 sbp}}, {{c1::end organ damage}}, and {{c1::lactate >…
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Septic shock is {{c1::severe sepsis}} with {{c2::hypotension}}, {{c2::end organ damage}}, and {{c2::lactate that doesn't respond to res…
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The blood glucose target range for patients in the ICU is {{c1::140}}-{{c1::180}}
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The qSOFA criteria is more {{c4::specific::specific or sensitive}} for sepsis and includes:1) {{c3::Altered mental status}}2) {{c2::BP <90}}3) {{c1…
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In suspected sepsis, the lactate is measured within {{c1::1 hour::time frame}}; if it is >{{c3::2}}, it should be remeasured within {{c2::2}}-{{c2:…
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Sepsis broad spectrum abx should include: - {{c4::Carbapenem}} - {{c3::Pip/tazo (Zosyn)}} - {{c2::Cefepime}} - {{c1::Vancomycin (i…
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Sepsis hypotension that is not responsive to fluids with a MAP of <65 should be started on {{c1::norepinephrine (Levophed)}}, if still not res…
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{{c2::Vasopressin}} is added to levophed if the patient is receiving the maximum dose of levophed (>{{c1::1}}-{{c1::1.5}} mg/kg)
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In septic and fluid-unresponsive patients, pressors should be added in the following order:1) Levophed2) {{c3::Vasopressin}}3) {{c2::Ep…
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When titrating septic patients off of pressors (levophed and vasopressin), titrate {{c1::levophed}} first
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The goal MAP with use of pressors is {{c2::65}} mmHg; the reason is higher goal MAPs (80-85) leads to a higher incidence of {{c1::A-fib}}
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{{c2::Phenylephrine}} is typically reserved as a last-line pressor or in patients who are very sensitive to {{c1::tachyarrhythmias}} from other presso…
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{{c3::Epinephrine::pressor}} can cause a type {{c2::II}} {{c1::lactic acidosis::pH disturbance}}
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{{c2::Vasopressin}} is typically added once {{c3::norepinephrine}} reaches {{c1::15}} mcg/min
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Dopamine has double the risk of {{c1::tachyarrhythmias}} compared to norepineprhine
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{{c2::Dopamine::pressor}} is reserved for patients in shock who are {{c1::bradycardic}}
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When weaning patients off of pressors, oral {{c1::midodrine}} may be helpful
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Are central lines required for pressors? {{c1::Preferred, but not required (don't delay giving pressors to get a central line)}}
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When administering pressors, a central line should be placed if giving {{c1::vasopressin}}
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Acute intra-abdominal inflammatory processes (e.g. free air) can present with a {{c2::sympathetic}} pleural effusion, which is {{c1::trans}}udative
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{{c2::Large}} bowel obstruction because of the location and also the {{c1::semi-c}}ircumferential {{c1::haustra}}
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{{c2::Small}} bowel obstruction because of the anatomic location and the {{c1::c}}ircumferential {{c1::valvulae}}
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Endotracheal tubes should be {{c1::4}}-{{c1::5}} cm above the {{c2::carina}}
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NG tube contraindications include {{c1::maxillofacial}} fracture
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A malpositioned central line in the azygous vein is treated with {{c1::leaving it alone}}
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On a lateral view chest x-ray, a malpositioned central line in the azygous vein will be seen migrating {{c1::pos}}teriorly
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{{c2::Nasopharyngeal}} airway size can be approximated using the distance from the patient's {{c1::nose}} to their {{c1::ear-lobe}}
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Stress steroids dosing for patients undergoing minor surgeries (e.g. hernia repair or biopsy) should receive {{c1::their usual morning dose (no supple…
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Stress steroid dosing for patients undergoing moderate surgical stress procedures (e.g. revascularization or joint replacement) should receive: -…
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Stress steroid drug of choice is {{c1::hydrocorti}}sone
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Stress steroid dosing for patients undergoing major surgical stress (e.g. esophagectomy, total proctocolectomy, or heart surgery) should receive:…
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